1. EPA weight-of evidence category, "not classifiable as to human carcinogenicity", usually due to inadequate data. US EPA, 2005. Office of Pesticide Programs. List of Chemicals Evaluated for Carcinogenic Potential. September 30, 2018. http://npic.orst.edu/chemicals_evaluated.pdf
2. US EPA Office of Pesticide Programs. List of Chemicals Evaluated for Carcinogenic Potential. September 30, 2018. http://npic.orst.edu/chemicals_evaluated.pdf
3. International Agency for Research on Cancer, World Health Organization (IARC) category, the agent (mixture) is possibly carcinogenic to humans. November 2, 2018. http://monographs.iarc.fr/ENG/Classification/index.php
4. Extension Toxicology Network (EXTOXNET) Pesticide Information Profiles. http://extoxnet.orst.edu/pips/ghindex.html
5. Illinois EPA, Endocrine Disruptors Strategy, February 1997. http://www.idaillinois.org/cdm/compoundobject/collection/edi/id/174979/rec/3
6. Northwest Coalition for Alternatives to Pesticides (NCAP), Pesticide Factsheets. http://www.pesticide.org/pesticide-factsheets.
7. Beyond Pesticides ChemWatch Factsheets. (Cited under factsheets on Beyond Pesticides Gateway)
8. US EPA, Office of Prevention, Pesticides and Toxic Substances, Reregistration Eligibility Decisions (REDs), Interim REDS (iREDs) and RED Factsheets. http://www.epa.gov/pesticides/reregistration/status.htm.
10. EPA weight-of-evidence category, "possible human carcinogen." US EPA, 2004. Office of Pesticide Programs. List of Chemicals Evaluated for Carcinogenic Potential. July 29, 2004. http://www.epa.gov/pesticides/carlist/
11. US EPA, 2000. Table 1: Toxicity Data by Category for Chemicals Listed under EPCRA Section 313. Toxic Release Inventory (TRI) Program. http://www.epa.gov/tri/trichemicals/hazardinfo/hazard_chronic_non-cancer95.pdf
12. EPA weight-of-evidence category, "Likely to be carcinogenic to humans (high dose); Not likely to be carcinogenic to humans (low doses)." US EPA, 2005. Office of Pesticide Programs. List of Chemicals Evaluated for Carcinogenic Potential. May 10, 2005. http://www.epa.gov/pesticides/carlist/
13. Frazier, L. and M.L. Hage. 2001. Reproductive Hazards of the Workplace. Europe: Wiley. Table 10: Partial List of Reproductive Toxins. https://web.archive.org/web/20100624221623/http://www.biosci.osu.edu/safety/CHP/Tables2001/Table10-11-00.pdf.
14. Environmental Defense Fund, Scorecard Database. http://www.scorecard.org/chemical-profiles/.
15. EPA weight-of-evidence category, "Group B2 – Probable Human Carcinogen." US EPA, 2005. Office of Pesticide Programs. List of Chemicals Evaluated for Carcinogenic Potential. May 10, 2005. http://www.epa.gov/pesticides/carlist/
16. EPA weight-of-evidence category, "Likely to be carcinogenic to humans." US EPA, 2005. Office of Pesticide Programs. List of Chemicals Evaluated for Carcinogenic Potential. May 10, 2005. http://www.epa.gov/pesticides/carlist/
17. New Jersey Department of Health and Senior Services, Right to Know Hazardous Substances Fact Sheets. Available online at http://web.doh.state.nj.us/rtkhsfs/indexfs.aspx
19. Tew, J.E. 1996. Protecting Honeybees from Pesticides. Ohio State University Cooperative Extension. http://web.archive.org/web/20031123075324/http://beelab.osu.edu/factsheets/sheets/2161.html
20. Briggs, S.A. 1992. Basic Guide to Pesticides: Their Characteristics and Hazards. Washington, DC: The Rachel Carson Council, 98. http://www.rachelcarsoncouncil.org/index.php?page=basic-guide-to-pesticides
21. California Environmental Protection Agency. Proposition 65: Chemicals Known to the State to Cause Cancer or Reproductive Toxicity. Office of Environmental Health Hazard Assessment. November 23, 2018.https://oehha.ca.gov/media/downloads/proposition-65//p65list112318.pdf
22. US EPA, 2006. Hazard Assessment of the Organophosphates. Hazard ID Committee Report. http://www.epa.gov/oppsrrd1/cumulative/2006-op/op_cra_main.pdf
23. US EPA, 1995. Monosodium Methanearsonate and Disodium Methanearsonate; Toxic Chemical Release Reporting; Community Right-to-Know. Federal Register Environmental Documents. https://www.govinfo.gov/app/details/FR-1995-04-20/95-9782.
24. US EPA. Integrated Risk Information System Database. http://www.epa.gov/iris/.
25. The Pesticide Management Education Program at Cornell University. Pesticide Active Ingredient Information. http://pmep.cce.cornell.edu/profiles/index.html.
26. EPA weight-of-evidence category, "Suggestive evidence of carcinogenicity but not sufficient to assess human carcinogenic potential." US EPA, 2005. Office of Pesticide Programs. List of Chemicals Evaluated for Carcinogenic Potential. May 10, 2005. http://www.epa.gov/pesticides/carlist/
27. National Library of Medicine. TOXNET Hazardous Substances Database. http://toxnet.nlm.nih.gov/cgi-bin/sis/htmlgen?HSDB.
28. Colborn, T., et al. 1994. Developmental Effects of Endocrine-Disrupting Chemicals in Wildlife and Humans. Environmental Impact Assessment Review 14:469-489. https://ehp.niehs.nih.gov/doi/10.1289/ehp.93101378.
29. Agency for Toxic Substances and Disease Registry. ToxFAQs. http://www.atsdr.cdc.gov/toxfaqs/index.asp.
30. Colborn, T., D. Dumanoski, and J.P. Myers. 1996. Our Stolen Future: Are We Threatening Our Fertility, Intelligence, and Survival? New York: Dutton. http://ourstolenfuture.org/Basics/chemlist.htm
31. Feldman, J. and T. Shistar. 1997. Poison Poles: A Report About their toxic trail and the safer alternatives. National Coalition Against the Misuse of Pesticides. https://www.beyondpesticides.org/programs/wood-preservatives/publications/poison-poles.
32. Department of Pesticide Regulation (DPR), Endosulfan- Risk Characterization Document. California Environmental Protection Agency, 2007. https://www.cdpr.ca.gov/docs/emon/pubs/tac/tacpdfs/endosulfan/endosulfan_sum.pdf.
33. Californians for Alternatives to Toxics (CATs). Toxicological Profiles. http://alt2tox.org/tox_profiles.htm.
34. Insecticide Resistance Action Committe (IRAC) eClassification of Chemical Mode of Action http://www.irac-online.org/eClassification/
35. Registry of Toxic Effects of Chemical Substances (RTECS). http://www.cdc.gov/niosh/rtecs/default.html
36. European Commission. Endocrine Disruptors: Study on Gathering Information on 435 Substances with Insufficient Data. Final Report. EU DG Environment: B4-3040/2001/325850/MAR/C2. BKH Consulting Engineers: M0355037. November 2002. http://ec.europa.eu/environment/chemicals/endocrine/pdf/bkh_report.pdf#page=76.
37. U.S. Geological Survey, Pesticides in the Nation's Streams and Ground Water, 1992-2001. http://water.usgs.gov/nawqa/pnsp/pubs/circ1291/appendix7.
38. Thurman, E.M. and A.E. Cromwell. 2000. Atmospheric Transport, Deposition, and Fate of Triazine Herbicides and Their Metabolites in Pristine Areas at Isle Royale National Park. Environmental Science and Technology 34:3079-3085. http://pubs.acs.org/doi/abs/10.1021/es000995l.
39. U.S. EPA, Office of Prevention, Pesticides and Toxic Substances, New Active Ingredients Factsheets:
40. Mineau, P., A. Baril, B.T. Collins , J. Duffe, G. Joerman, R. Luttik. 2001. Reference values for comparing the acute toxicity of pesticides to birds. Reviews of Environmental Contamination and Toxicology 170:13-74. http://web.archive.org/web/20081006213641/http://www.abcbirds.org/abcprograms/policy/pesticides/aims/aims/toxicitytable.cfm
41. Arctic Monitoring and Assessment Programme. 2009. AMAP Assessment 2009: Human Health in the Arctic. https://www.amap.no/documents/doc/amap-assessment-2009-human-health-in-the-arctic/98.
42. Hageman, et al. 2006. Atmospheric Deposition of Current-Use and Historic-Use Pesticides in Snow at National Parks in the Western United States. Environ. Sci. Technol., 2006, 40 (10), pp 3174–3180. http://pubs.acs.org/doi/abs/10.1021/es060157c
43. Pesticide Action Network Pesticide Database. http://www.pesticideinfo.org/Search_Chemicals.jsp.
44. Federal Register. September 5, 2008. http://www.epa.gov/fedrgstr/EPA-PEST/2008/September/Day-05/p20548.htm
45. Federal Register. March 21, 2003. http://www.epa.gov/fedrgstr/EPA-PEST/2003/March/Day-21/p6822.htm
46. Fluoride Action Alert Pesticide Project Factsheets. http://www.fluoridealert.org/f-pesticides.htm
47. EPA docket ID EPA-HQ-OPP-2010-0324. August 17, 2011. https://www.regulations.gov/docket?D=EPA-HQ-OPP-2010-0324.
48. USDA/Forest Service. Dinotefuran: Human Health and Ecological Risk Assessment Final Report. April 24, 2009. http://www.fs.fed.us/foresthealth/pesticide/pdfs/0521803b_Dinotefuran.pdf.
49. U.S. Department of Energy Bonneville Power Administration. 2000. Halosulfuron: Herbicide Fact Sheet https://www.bpa.gov/efw/Analysis/NEPADocuments/nepa/Vegetation_Management/sheets/Halosulfuron.pdf.
50.IUPAC Agrochemical Information. http://sitem.herts.ac.uk/aeru/iupac/
51. Federal Register. Fenbuconazole Pesticide Tolerance. January 15, 2002. http://www.epa.gov/fedrgstr/EPA-PEST/2003/March/Day-21/p6822.htm.
52. California Department of Pesticide Regulation, Public Reports on New Active Ingredients http://www.cdpr.ca.gov/docs/registration/ais/publicreports/publicreports.htm
53. EPA Pesticide Registration Review Status http://www.epa.gov/oppsrrd1/registration_review/reg_review_status.htm
54. National Toxiocology Program. 14th Report on Carcinogens. Nov 3, 2016. https://ntp.niehs.nih.gov/pubhealth/roc/index-1.html.
55. University of California Statewide Integrated Pest Management Program. Pesticide Information. http://www.ipm.ucdavis.edu/GENERAL/pesticides.html
56. PAN Pesticide Database. http://www.pesticideinfo.org/Search_Chemicals.jsp
57. Yueh, MF et al. 2014. The commonly used antimicrobial additive triclosan is a liver tumor promoter. PNAS doi: 10.1073/pnas.141911911. Triclosan promotes liver cancer cell development and proliferation in mice through pathways common to humans.
58. Kim, J et al. 2017. Triclosan affects axon formation in the neural development stages of zebrafish embryos (Danio rerio). Environmental Pollution doi: 10.1016/j.enjvpol.2017.12.110.
59. Lassen et al. 2016. Prenatal Triclosan Exposure and Anthropometric Measures Including Anogenital Distance in Danish Infants. Environmental Health Perspectives doi: 10.1289/ehp.1409637. Prenatal triclosan exposure associated with reduced head circumference, a trait linked to cognitive impairment.
60. Stuart, M et al. 2012. Review of risk from potential emerging contaminants in UK groundwater. Science of the Total Environment 416, 1-21. UK Environment Agency detected triclosan in groundwater 22 times in 22 sites over the period 1992-2009, at a maximum concentration of 2.11 µg/L.
61. Karnjanapiboonwong, A et al. 2011. Occurrence of PPCPs at a Wastewater Treatment Plant and in Soil and Groundwater at a Land Application Site. Water, Air, & Soil Pollution 216(1-4), 257-273. Triclosan detected in 5 out of 7 groundwater samples from a West Texas Land Application Site, at concentrations ranging 12-53 ng/L.
62. Parenti, CC et al. 2018. Environmental concentrations of triclosan activate cellular defence mechanism and generate cytotoxicity on zebrafish (Danio rerio) embryos. Science of the Total Environment 650, 1752-1758. Triclosan levels commonly found in the environment invoke oxidative stress immune responses and cause high levels of cell death in zebrafish embryos.
63. Lee, HR et al. 2014. Progression of Breast Cancer Cells Was Enhanced by Endocrine-Disrupting Chemicals, Triclosan and Octylphenol, via an Estrogen Receptor-Dependent Signaling Pathway in Cellular and Mouse Xenograft Models. Chemical Research in Toxicology doi: 10.1021/tx5000156.
64. Riad, M et al. 2017. Reproductive toxic impact of subchronic treatment with combined butylparaben and triclosan in weanling male rats. J Biochem Mol Toxicol doi: 10.1002/jbt.22037. Treatment with triclosan alone causes testicular oxidative stress and DNA damage, leading to a marked reduction in sperm count and sperm motility.
65. Jurewicz, J et al. 2017. Environmental levels of triclosan and male fertility. Environmental Science and Pollution Research 25(6), 5484-5490. Men with higher urinary concentrations of triclosan have poorer semen quality, exhibiting a greater percentage of sperm with abnormal morphology as compared to men with lower triclosan levels.
66. Kang, D. et al., 2008. Cancer incidence among pesticide applicators exposed to trifluralin in the Agricultural Health Study. Environmental Research, 107(2), 271-276. Regression analysis of pesticide exposures and cancer incidence across a cohort of 50,127 private and commercial pesticide applicators show that above-average levels of trifularlin exposure significantly predict incidence of colon cancer, controlling for lifestyle factors and other agricultural exposures.
67. Kılıç, Z.S., Aydın, S., Bucurgat, Ü.Ü. and Başaran, N., 2018. In vitro genotoxicity assessment of dinitroaniline herbicides pendimethalin and trifluralin. Food and Chemical Toxicology, 113, 90-98. Trifluralin exposure at concentrations as low as 1.7 ppb causes significant damage to DNA and chromosomes in human peripheral lymphocytes, demonstrating genotoxicity as a mechanism of carcinogenicity.
68. Saghir, S.A., Charles, G.D., Bartels, M.J., Kan, L.H., Dryzga, M.D., Brzak, K.A. and Clark, A.J., 2008. Mechanism of trifluralin-induced thyroid tumors in rats. Toxicology Letters, 180(1) 38-45. Trifluralin treatment increased the conjugation and excretion of thyroid hormones (TH), thereby increasing pituitary production of thyroid stimulating hormone (TSH) and causing thyroid tumor development. The mode of action for tumor promotion by trifluralin has relevance to human health, as increased bile excretion coupled with decreased functioning of a shared class of thyroid hormone binding agents would be expected to cause similar effects in humans.
69. Emmerson, J.L., Pierce, E.C., McGrath, J.P., 1980. The chronic toxicity of compound 36352 (trifluralin) given as a compound of the diet to the Fischer 344 rats for two years. StudiesR-87 andR-97 (Elanco Products Co., Division of Eli Lilly and Co., Indianapolis, IN). Cited in Reregistration Eligibility Decision (RED) on trifluralin, USEPA, Office of Prevention, Pesticides and Toxic Substances. EPA 738-R-95-040, April 1996. Chronic exposure to trifluralin causes thyroid tumor development in rats.
70. Zhang, L., Rana, I., Taioli, E., Shaffer, R.M. and Sheppard, L., 2019. Exposure to Glyphosate-Based Herbicides and Risk for Non-Hodgkin Lymphoma: A Meta-Analysis and Supporting Evidence. Mutation Research/Reviews in Mutation Research. Meta-analysis of every available published human study on NHL and glyphosate, including the most recently updated data from the ongoing U.S. Agricultural Health Study, published in 2018. Statistical analysis revealed a 41% increased risk of NHL resulting from high exposure to glyphosate-based herbicide.
71. EFSA, 2018. Peer review of the pesticide risk assessment of the active substance variant florpyrauxifen-benzyl: https://efsa.onlinelibrary.wiley.com/doi/epdf/10.2903/j.efsa.2018.5378.
72. Khanam, S., 2017. Effect of Carbaryl on Hemoglobin and Hematocrit Values of Broiler Chicks. Malaysian Journal of Medical Research, 1(2), pp.38-40. http://ejournal.lucp.net/index.php/mjmr/article/view/139
73. Hussain R, Ali F, Rafique A, Ghaffar A, Jabeen G, Rafay M, Liaqat S, Khan I, Malik R, Khan MK, Niaz M, Akram K and Masood A, 2019. Exposure to sub-acute concentrations of glyphosate induce clinicohematological, serum biochemical and genotoxic damage in adult cockerels. Pak Vet J, 39(2): 181-186. http://dx.doi.org/10.29261/pakvetj/2019.064
74. Neto da Silva, K., Garbin Cappellaro, L., Ueda, C.N., Rodrigues, L., Pertile Remor, A., Martins, R.D.P., Latini, A. and Glaser, V., 2020. Glyphosate-based herbicide impairs energy metabolism and increases autophagy in C6 astroglioma cell line. Journal of Toxicology and Environmental Health, Part A, pp.1-15. https://www.tandfonline.com/doi/abs/10.1080/15287394.2020.1731897
75. Rappazzo, K.M., Warren, J.L., Davalos, A.D., Meyer, R.E., Sanders, A.P., Brownstein, N.C. and Luben, T.J., 2019. Maternal residential exposure to specific agricultural pesticide active ingredients and birth defects in a 2003–2005 North Carolina birth cohort. Birth defects research, 111(6), pp.312-323. https://onlinelibrary.wiley.com/doi/epdf/10.1002/bdr2.1448
76. Ledoux, M.L., Hettiarachchy, N., Yu, X., Howard, L. and Lee, S.O., 2019. Penetration of glyphosate into the food supply and the incidental impact on the honey supply and bees. Food Control, p.106859. https://doi.org/10.1016/j.foodcont.2019.106859
77. Zgurzynski, M.I. and Lushington, G.H., 2019. Glyphosate Impact on Apis mellifera Navigation: A Combined Behavioral and Cheminformatics Study. EC Pharmacology and Toxicology, 7, pp.806-824. https://www.ecronicon.com/ecpt/pdf/ECPT-07-00336.pdf
78. Rendón-von Osten, J. and Dzul-Caamal, R., 2017. Glyphosate residues in groundwater, drinking water and urine of subsistence farmers from intensive agriculture localities: a survey in Hopelchén, Campeche, Mexico. International journal of environmental research and public health, 14(6), p.595. https://www.mdpi.com/1660-4601/14/6/595/htm
80. Office of Prevention, Pesticides and Toxic Substances, 1998. Memorandum - Isoxaflutole. Environmental Protection Agency. https://www3.epa.gov/pesticides/chem_search/cleared_reviews/csr_PC-123000_5-Feb-98_a.pdf
81.Reuter, W., 2019. Toxicology of glyphosate, isoxaflutole, dicamba and possible combination effects. Testbiotech.www.testbiotech.org/sites/default/files/Tox_Evaluation _Glyphosate _Dicamba_Isoxaflutole.pdf. Accessed 2020.
82. Mesnage, R., Biserni, M., Wozniak, E., Xenakis, T., Mein, C.A. and Antoniou, M.N., 2018. Comparison of transcriptome responses to glyphosate, isoxaflutole, quizalofop-p-ethyl and mesotrione in the HepaRG cell line. Toxicology reports, 5, pp.819-826. https://doi.org/10.1016/j.toxrep.2018.08.005
83. Melo, C.A.D., Medeiros, W.N., Santos, L.T., Ferreira, F.A., Tiburcio, R.A.S. and Ferreira, L.R., 2010. Leaching of sulfentrazone, isoxaflutole and oxyfluorfen in three soil profiles. Planta Daninha, 28(2), pp.385-392. 10.1590/S0100-83582010000200018
84. Hu, Y., Zhang, Y., Vinturache, A., Wang, Y., Shi, R., Chen, L., Qin, K., Tian, Y. and Gao, Y., 2020. Effects of environmental pyrethroids exposure on semen quality in reproductive-age men in Shanghai, China. Chemosphere, 245, p.125580. https://doi.org/10.1016/j.chemosphere.2019.125580
85. Walsh, E.M., Sweet, S., Knap, A., Ing, N. and Rangel, J., 2020. Queen honey bee (Apis mellifera) pheromone and reproductive behavior are affected by pesticide exposure during development. Behavioral Ecology and Sociobiology, 74(3), pp.1-14. https://doi.org/10.1007/s00265-020-2810-9
86. Park, A.S., Ritz, B., Yu, F., Cockburn, M. and Heck, J.E., 2020. Prenatal pesticide exposure and childhood leukemia–A California statewide case-control study. International journal of hygiene and environmental health, 226, p.113486. DOI: 10.1016/j.ijheh.2020.113486
87. Anselmi, L., Bove, C., Coleman, F.H., Le, K., Subramanian, M.P., Venkiteswaran, K., Subramanian, T. and Travagli, R.A., 2018. Ingestion of subthreshold doses of environmental toxins induces ascending Parkinsonism in the rat. npj Parkinson's Disease, 4(1), pp.1-10. DOI: 10.1038/s41531-018-0066-0
88. Brouwer, M., Huss, A., van der Mark, M., Nijssen, P.C., Mulleners, W.M., Sas, A.M., Van Laar, T., de Snoo, G.R., Kromhout, H. and Vermeulen, R.C., 2017. Environmental exposure to pesticides and the risk of Parkinson's disease in the Netherlands. Environment international, 107, pp.100-110. DOI: 10.1016/j.envint.2017.07.001
89. Hou, L., Zhang, C., Wang, K., Liu, X., Wang, H., Che, Y., Sun, F., Zhou, X., Zhao, X. and Wang, Q., 2017. Paraquat and maneb co-exposure induces noradrenergic locus coeruleus neurodegeneration through NADPH oxidase-mediated microglial activation. Toxicology, 380, pp.1-10. DOI: 10.1016/j.tox.2017.02.009
90. de Mattos, I.M., Soares, A.E. and Tarpy, D.R., 2018. Mitigating effects of pollen during paraquat exposure on gene expression and pathogen prevalence in Apis mellifera L. Ecotoxicology, 27(1), pp.32-44.
91. Badroo, I.A., Wani, K.A., Nandurkar, H.P. and Khanday, A.H., 2019. Renewal Acute Toxicity of Broad-Spectrum Herbicide, Paraquat Dichloride in Channa punctatus (Bloch). Environmental Claims Journal, 31(4), pp.289-303. DOI: 10.1080/10406026.2019.1609796
92. Ayanda, O.I., Oniye, S.J. and Auta, J.A., 2017. Behavioural and Some Physiological Assessment of Glyphosate and Paraquat Toxicity to Juveniles of African Catfish, Clarias gariepinus. pakistan Journal of Zoology, 49(1), pp.183-190. DOI: 10.17582/journal.pjz/2017.49.1.83.190
93. Moustakas, M., Malea, P., Zafeirakoglou, A. and Sperdouli, I., 2016. Photochemical changes and oxidative damage in the aquatic macrophyte Cymodocea nodosa exposed to paraquat-induced oxidative stress. Pesticide biochemistry and physiology, 126, pp.28-34. DOI: 10.1016/j.pestbp.2015.07.003
96. Hojo, Y., Shiraki, A., Tsuchiya, T., Shimamoto, K., Ishii, Y., Suzuki, K., Shibutani, M. and Mitsumori, K., 2012. Liver tumor promoting effect of etofenprox in rats and its possible mechanism of action. The Journal of toxicological sciences, 37(2), pp.297-306. https://doi.org/10.2131/jts.37.297
97. Benli, A.C.K., 2015. The influence of etofenprox on narrow clawed crayfish (Astacus leptodactylus Eschscholtz, 1823): Acute toxicity and sublethal effects on histology, hemolymph parameters, and total hemocyte counts. Environmental toxicology, 30(8), pp.887-894. https://onlinelibrary.wiley.com/doi/abs/10.1002/tox.21963
98. Pesticide Action Network, 2019. PAN Pesticide Database. http://www.pesticideinfo.org/Detail_Chemical.jsp?Rec_Id=PRI3067
99. De Coster, S. and Van Larebeke, N., 2012. Endocrine-disrupting chemicals: associated disorders and mechanisms of action. Journal of environmental and public health, 2012. https://doi.org/10.1155/2012/713696
100. Hayashi, K., Nakae, A., Fukushima, Y., Sakamoto, K., Furuichi, T., Kitahara, K., Miyazaki, Y., Ikenoue, C., Matumoto, S. and Toda, T., 2010. Contamination of rice by etofenprox, diethylphthalate and alkylphenols: effects on first delivery and sperm count in mice. The Journal of toxicological sciences, 35(1), pp.49-55. https://doi.org/10.2131/jts.35.49
101. Terzaghi, E., Vitale, C.M. and Di Guardo, A., 2020. Modelling peak exposure of pesticides in terrestrial and aquatic ecosystems: importance of dissolved organic carbon and vertical particle movement in soil. SAR and QSAR in Environmental Research, 31(1), pp.19-32. https://doi.org/10.1080/1062936X.2019.1686715
102. Merola, V. and Dunayer, E., 2006. The 10 most common toxicoses in cats. VETERINARY MEDICINE-BONNER SPRINGS THEN EDWARDSVILLE-, 101(6), p.339. https://www.aspcapro.org/sites/default/files/zl-vetm0606_339-342.pdf
104. Mossa, A.T.H., Refaie, A.A., Ramadan, A. and Bouajila, J., 2013. Antimutagenic effect of Origanum majorana L. essential oil against prallethrin-induced genotoxic damage in rat bone marrow cells. Journal of medicinal food, 16(12), pp.1101-1107. https://doi.org/10.1089/jmf.2013.0006
105. Bhaskar, E.M., Moorthy, S., Ganeshwala, G. and Abraham, G., 2010. Cardiac conduction disturbance due to prallethrin (pyrethroid) poisoning. Journal of Medical Toxicology, 6(1), pp.27-30. https://doi.org/10.1007/s13181-010-0032-7
106. Alam, Z., Mohsin, A., Yunus, S.M., Ahmad, F. and Faruqi, N.A., 2017. EFFECT OF PRALLETHRIN VAPOURS ON CEREBELLAR CORTEX OF ALBINO RATS: A NEUROHISTOLOGICAL STUDY. JOURNAL OF ANATOMICAL SCIENCES, 25(1), pp.8-11. http://www.asiup.in/journals/june-2017/JAS%20JOURNAL%202017.pdf#page=15
107. Botnariu, G., Birsan, C., Podoleanu, C., Moldovan, C., Stolnicu, S. and Chiriac, A., 2016. Skin necrosis caused by prallethrin—A worldwide used insecticide. Environmental toxicology and pharmacology, 43, pp.103-104. https://doi.org/10.1016/j.etap.2016.03.002
108. Na, H.G., Kim, Y.D., Choi, Y.S., Bae, C.H. and Song, S.Y., 2018. Allethrin and prallethrin stimulates MUC5AC expression through oxidative stress in human airway epithelial cells. Biochemical and biophysical research communications, 503(1), pp.316-322. https://doi.org/10.1016/j.bbrc.2018.06.022
109. Mossa, A.T.H., Refaie, A.A., Ramadan, A. and Bouajila, J., 2013. Amelioration of prallethrin-induced oxidative stress and hepatotoxicity in rat by the administration of Origanum majorana essential oil. BioMed research international, 2013. doi: 10.1155/2013/859085
110. Lerro, C.C., Hofmann, J.N., Andreotti, G., Koutros, S., Parks, C.G., Blair, A., Albert, P.S., Lubin, J.H., Sandler, D.P. and Beane Freeman, L.E., 2020. Dicamba use and cancer incidence in the agricultural health study: an updated analysis. International Journal of Epidemiology.https://doi.org/10.1093/ije/dyaa066
111.Herrero-Hernández, E., Simón-Egea, A.B., Sánchez-Martín, M.J., Rodríguez-Cruz, M.S. and Andrades, M.S., 2020. Monitoring and environmental risk assessment of pesticide residues and some of their degradation products in natural waters of the Spanish vineyard region included in the denomination of origin jumilla. Environmental Pollution, p.114666. https://doi.org/10.1016/j.envpol.2020.114666
112. Stoker, T.E. and Kavlock, R.J., 2010. Pesticides as endocrine-disrupting chemicals. In Hayes' Handbook of Pesticide Toxicology (pp. 551-569). Academic Press. https://doi.org/10.1016/B978-0-12-374367-1.00018-5
113. Knudsen, T.B., Martin, M.T., Kavlock, R.J., Judson, R.S., Dix, D.J. and Singh, A.V., 2009. Profiling the activity of environmental chemicals in prenatal developmental toxicity studies using the US EPA's ToxRefDB. Reproductive toxicology, 28(2), pp.209-219. https://doi.org/10.1016/j.reprotox.2009.03.016
114. Ran, D., Wu, X., Zheng, J., Yang, J., Zhou, H., Zhang, M. and Tang, Y., 2007. Study on the interaction between florasulam and bovine serum albumin. Journal of fluorescence, 17(6), pp.721-726. https://doi.org/10.1007/s10895-007-0226-9
115. US EPA, Office of Prevention, Pesticides and Toxic Substances, Science Data Evaluation. https://iaspub.epa.gov/apex/pesticides/florasulam
116. The International Union of Pure and Applied Chemistry (IUPAC), Pesticide Properties Database (PPDB), florasulam (Ref: DE 570). https://sitem.herts.ac.uk/aeru/iupac/Reports/322.htm
117. The Dow Chemical Company, Product Safety Assessment, Florasulam. http://msdssearch.dow.com/PublishedLiteratureDOWCOM/dh_07cf/0901b803807cfdd2.pdf?filepath=productsafety/pdfs/noreg/233-00436.pdf&fromPage=GetDoc
118. Hernández-Borges, J., García-Montelongo, F.J., Cifuentes, A. and Rodríguez-Delgado, M.Á., 2005. Determination of herbicides in mineral and stagnant waters at ng/L levels using capillary electrophoresis and UV detection combined with solid-phase extraction and sample stacking. Journal of Chromatography A, 1070(1-2), pp.171-177. https://doi.org/10.1016/j.chroma.2005.02.053
119. Weber, G., Christmann, N., Thiery, A.C., Martens, D. and Kubiniok, J., 2018. Pesticides in agricultural headwater streams in southwestern Germany and effects on macroinvertebrate populations. Science of the Total Environment, 619, pp.638-648. https://doi.org/10.1016/j.scitotenv.2017.11.155
120. Sandin, M., Piikki, K., Jarvis, N., Larsbo, M., Bishop, K. and Kreuger, J., 2018. Spatial and temporal patterns of pesticide concentrations in streamflow, drainage and runoff in a small Swedish agricultural catchment. Science of the Total Environment, 610, pp.623-634. https://doi.org/10.1016/j.scitotenv.2017.08.068
121. US EPA, Office of Prevention, Pesticides and Toxic Substances, Pesticide Fact Sheet. https://www3.epa.gov/pesticides/chem_search/reg_actions/registration/fs_PC-129171_22-May-97.pdf
122. Wisconsin Department of Natural Resources, 2012. Imazamox Chemical Fact Sheet. https://dnr.wi.gov/lakes/plants/factsheets/ImazamoxFactsheet.pdf
123. Kaur, P., Kaur, P. and Kaur, K., 2020. Adsorptive removal of imazethapyr and imazamox from aqueous solution using modified rice husk. Journal of Cleaner Production, 244, p.118699. https://doi.org/10.1016/j.jclepro.2019.118699
124. Demirci, Ö., Toptancı, B.Ç. and Kızıl, M., 2016. In Vitro Studies on Pesticide-Induced Oxidative DNA Damage. Journal of the Turkish Chemical Society Section A: Chemistry, 3(3), pp.479-490. https://www.researchgate.net/profile/Oezlem_Demirci/publication/308977224_In_Vitro_Studies_on_Pesticide-Induced_Oxidative_DNA_Damage/links/58bf06a7aca272bd2a3acf3a/In-Vitro-Studies-on-Pesticide-Induced-Oxidative-DNA-Damage.pdf
125. Di Marzio, W.D., Cifoni, M., Sáenz, M.E., Galassi, D.M. and Di Lorenzo, T., 2018. The ecotoxicity of binary mixtures of Imazamox and ionized ammonia on freshwater copepods: Implications for environmental risk assessment in groundwater bodies. Ecotoxicology and Environmental Safety, 149, pp.72-79. https://www.sciencedirect.com/science/article/pii/S0147651317307844
126. Tsatsakis, A., Tyshko, N.V., Docea, A.O., Shestakova, S.I., Sidorova, Y.S., Petrov, N.A., Zlatian, O., Mach, M., Hartung, T. and Tutelyan, V.A., 2019. The effect of chronic vitamin deficiency and long term very low dose exposure to 6 pesticides mixture on neurological outcomes–A real-life risk simulation approach. Toxicology letters, 315, pp.96-106. https://www.sciencedirect.com/science/article/pii/S0378427419302140
127. Taghizadehghalehjoughi, A. and Cicek, B., 2018. Momordica and Pycnogenol Can Tolerate Imazamox Induced Toxicity in L929 Cells Line: In Vitro Study. In Multidisciplinary Digital Publishing Institute Proceedings (Vol. 2, No. 25, p. 1584). https://doi.org/10.3390/proceedings2251584
128. Sevim, Ç., Çomaklı, S., Taghizadehghalehjoughi, A., Özkaraca, M., Mesnage, R., Kovatsi, L., Burykina, T.I., Kalogeraki, A., Antoniou, M.N. and Tsatsakis, A., 2019. An imazamox-based herbicide causes apoptotic changes in rat liver and pancreas. Toxicology reports, 6, pp.42-50. https://doi.org/10.1016/j.toxrep.2018.11.00
129. SEVĠM, A.G.Ç. and ÇOMAKLI, Ö.Ü.S., HĠSTOPATHOLOGĠCAL CHANGES OF THE TESTES ĠN COMMONLY USED HERBĠCĠDES (GLUFOSĠNATE AND IMAZAMOX) EXPOSURE. https://www.researchgate.net/profile/Cigdem_Sevim/publication/330214617_HISTOPATHOLOGICAL_CHANGES_OF_THE_TESTES_ON_COMMONLY_USED_HERBICIDES_GLUFOSINATE_AND_IMAZAMOX_EXPOSURE/links/5c346f0792851c22a3639eb0/HISTOPATHOLOGICAL-CHANGES-OF-THE-TESTES-ON-COMMONLY-USED-HERBICIDES-GLUFOSINATE-AND-IMAZAMOX-EXPOSURE.pdf